Neurological Deficits in Congenital Kyphosis

Abstract from the SRS 2001 Annual Meeting
Alexandre Y. Mushkin, M.D.
Robert B. Winter, M.D.
John E. Lonstein, M.D.
Francis Denis, M.D.

INTRODUCTION:
Neurological deficits are a known complication of congenital kyphosis. The role of the type of anomaly is documented, but the role of canal diameter is not documented.

PURPOSE:
To investigate the relationship between the anomaly and canal size in congenital kyphosis and the occurrence of neurological deficits.

METHODS:
A retrospective chart and radiographic review was performed on all cases of congenital kyphosis seen. The canal diameter narrowing was expressed as the percent of the adjacent normal level, where a is the antero posterior (AP) size of the canal at the level above the apex of the kyphosis and b is the AP size of the canal at the apex of the kyphosis.

RESULTS:
Twelve of the 61 cases had neurological loss due to the kyphosis, ranging from reflex changes to paraparesis. The Type 1 kyphosis was divided into three subcategories - complete vertebral agenesis; a rudimentary vertebra (hypogenesis) and a wedged vertebra (hypoplasia).

  N Neuro-Loss Kyphosis Stenosis
Type I. Agenesis 6
4 (67%)
59°
53%
Type I. Hypogenesis 7
3 (43%)
47°
37%
Type I. Hypoplasia 13
3 (23%)
41°
13%
Type II 27
0
46°
2%
Type III 8
2 (25%)
58°
20%

The average stenosis increased sharply if the kyphosis was >50°, with kyphosis of 60° being accompanied by canal narrowing of 50%. In addition the number of cases of neurological loss increased if the kyphosis was >60°.

SUMMARY:
The occurrence of neurological loss in congenital kyphosis is related to the type of anomaly, the degree of kyphosis and the canal stenosis at the apex of the deformity.

Last Updated: 08/10/2007